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Plasma prekallikrein Plasma kallikrein Coagulation factor XIIa Coagulation factor XIIa Coagulation factor XII Collagen alpha-1(I) chain Coagulation factor VII Tissue factor Tissue factor Fibrin (loose) Fibrin (loose) Fibrin (loose) Fibrin (mesh) Coagulation factor XIII A chain Coagulation factor XIIIa Coagulation factor XIII B chain Coagulation factor XI Coagulation factor XIa Coagulation factor IX Prothrombin Coagulation factor VIII Coagulation factor V Coagulation factor VIII Coagulation factor IX Coagulation factor X Coagulation factor V Coagulation factor X Prothrombin Thrombin Fibrinogen alpha chain Fibrinogen beta chain Fibrinogen gamma chain Tissue-type plasminogen activator Plasminogen Fibrin degradation products Plasmin Coagulation factor X Coagulation factor VIIa Tranexamic Acid Cortexolone Calcium Calcium Calcium Calcium Calcium Cortexolone Calcium Calcium Calcium Calcium Tenase Complex Prothrombinase Complex Intrinsic Pathway Extrinsic Pathway Vascular Injury At the site of injury collagen and tissue factor are exposed to coagulation factors in the blood activating the extrinsic and intrinsic pathways. Extracellular Space The fibrin mesh/clot forms at the site of vascular injury, blocking the leaking blood. ranexamic acid competitively and reversibly inhibits the activation of plasminogen via binding at several distinct sites, including four or five low-affinity sites and one high-affinity site Plasmin is unable to break down the fibrin clot. This prevents dissolution of fibrin, thereby stabilizing the clot and preventing hemorrhage. Thrombin converts fibrinogen to an unstable fibrin. Activated factor XIIIa strengthens the fibrin-fibrin links creating a stable fibrin mesh which forms the clot
KLKB1 KLKB1 F12 F12 F12 COL1A1 F7 Unknown Unknown Unknown Unknown FGG FGG F13A1 Unknown Unknown Unknown Unknown F9 Unknown F8 F5 F8 F9 F10 F5 F10 Unknown Unknown Unknown Unknown FGG PLAT PLG FGG PLG F10 F7 Tranexamic Acid
KLKB1 KLKB1 F12 F12 F12 COL1A1 F7 FGG FGG F13A1 F9 F8 F5 F8 F9 F10 F5 F10 FGG PLAT PLG FGG PLG F10 F7 TrnxmcA Cortexo Ca2+ Ca2+ Ca2+ Ca2+ Ca2+ Cortexo Ca2+ Ca2+ Ca2+ Ca2+ Tenase Complex Prothrombinase Complex Intrinsic Pathway Extrinsic Pathway Vascular Injury At the site of injury collagen and tissue factor are exposed to coagulation factors in the blood activating the extrinsic and intrinsic pathways. Extracellular Space The fibrin mesh/clot forms at the site of vascular injury, blocking the leaking blood. ranexamic acid competitively and reversibly inhibits the activation of plasminogen via binding at several distinct sites, including four or five low-affinity sites and one high-affinity site Plasmin is unable to break down the fibrin clot. This prevents dissolution of fibrin, thereby stabilizing the clot and preventing hemorrhage. Thrombin converts fibrinogen to an unstable fibrin. Activated factor XIIIa strengthens the fibrin-fibrin links creating a stable fibrin mesh which forms the clot
KLKB1 KLKB1 F12 F12 F12 COL1A1 F7 FGG FGG F13A1 F9 F8 F5 F8 F9 F10 F5 F10 FGG PLAT PLG FGG PLG F10 F7 TrnxmcA